closed-circuit cardiotome
cardiotome with closed reservoir

Я не нашел других терминов для "кардиотом" в английском языке, но увидел все же cardiotome. Я так догадываюсь, что это некий контур (circuit) или резервуар, по которому или через который происходит циркуляция крови.

Что-то напоминаюет такое:

A perfusion system according to any preceding claim, further comprising a control system (10), said control system comprising:
at least one clamp (82, 84, 88) on a line of the perfusion system, the clamp being capable of opening and closing so that when the clamp is open, fluid may flow through the line and when the clamp is closed, fluid is prevent from flowing through the line; and
means (102) for controlling the clamp(s).

https://patents.google.com/patent/EP0808185B1/en

The closed-circuit design without cardiotomy suction andvents eliminates any air-fluid interface, and pericardialshed blood is not reinfused. Several studies comparingMECC circuits to crude cardiopulmonary bypass circuitshave found an attenuated systemic inflammatoryresponse [38], reduced microembolisation [39] andreduced the need for red cell transfusion [40] associatedwith the former. It is likely that the elimination of cardiot-omy suction contributed significantly to the improvedoutcome of these patients, and further studies are requiredto compare MECC directly to CPB without cardiotomysuction

Pericardial shed bloodBlood which has extravasated into the pericardial or pleu-ral cavities and which is subsequently aspirated by cardi-otomy suction differs markedly from intravascular bloodor blood within a closed CPB circuit. Surgical trauma fromopening the chest results in substantial tissue damage andrelease of tissue factor [9,10]. Exposure of blood to tissuefactor results in rapid activation of the extrinsic pathwayof the coagulation system with release of thrombin andfibrin. In addition, tissue plasminogen activator releasestimulates fibrinolysis. Analysis of pericardial shed bloodshows high concentrations of markers of clotting activa-Published: 25 October 2007Journal of Cardiothoracic Surgery 2007, 2:46 doi:10.1186/1749-8090-2-46Received: 30 May 2007Accepted: 25 October 2007This article is available from: http://www.cardiothoracicsurgery.org/content/2/1/46© 2007 Lau et al; licensee BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/2.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
Journal of Cardiothoracic Surgery 2007, 2:46 http://www.cardiothoracicsurgery.org/content/2/1/46Page 2 of 5(page number not for citation purposes)tion and fibrinolysis [11,12] and a low heparin concentra-tion [7]. Activation of the coagulation cascade inevitablyresults in activation of the other inflammatory cascades.High levels of inflammatory markers such as TNF-alpha,IL-6, IL-8 and have been identified in pericardial shedblood [8,13,14].

https://www.researchgate.net/publication/5885096_Coronary_ar...

Abstract

In 20 patients undergoing coronary artery bypass grafting, we studied prospectively systemic blood activation, blood loss, and the need for donor blood when using an extracorporeal circuit equipped at random with one of two different venous reservoirs. In 10 patients we used an open venous reservoir system (ORS) consisting of a hard shell venous reservoir with an integral cardiotomy filter, and in 10 patients we used a closed reservoir system consisting of a collapsible venous reservoir and separate cardiotomy reservoir. Concentrations of complement 3a, elastase, thromboxane B2, and fibrin degradation products showed a biphasic course, especially in ORS patients. During bypass, we observed a first peak of levels of complement 3a, thromboxane B2, fibrin degradation products, and elastase, which was higher in ORS patients than in patients with the closed system, because their blood continuously contacted the foreign materials of the filter and air in the open reservoir, which was avoided in the closed reservoir. Intensive blood-foreign material contact also caused the highest (p < 0.05) hemolysis in ORS patients. The larger amount of hemolytic products in ORS patients theoretically resulted in a temporary decrease in capacity of their Kupffer cells to clear endotoxin released after aortic declamping. This theory might explain the significantly (p < 0.01) higher second peak of activated products after declamping that was observed in ORS patients. Due to increased blood activation, the largest (p < 0.001) amount of shed blood loss, greatest (p < 0.05) need for colloid-crystalloid infusion, and largest (not significant) need for donor blood were found in ORS patients (0.8 +/- 0.4 versus 0.2 +/- 0.2 units of packed cells).(ABSTRACT TRUNCATED AT 250 WORDS)

https://pubmed.ncbi.nlm.nih.gov/7771838/